Nutrient-responsive dermatoses

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The skin is a large organ, and 25–30% of an animal’s daily protein intake is required for optimal hair growth, cornification and formation of a functional epidermal barrier. Essential fatty acids (EFAs), vitamin A, zinc and copper are all known to play a major role in homeostasis of the skin. Owing to the excellence of current commercial diets, true nutrient deficiencies are rarely seen in companion animals and most of the diseases discussed here are characterized as . However, the quality of the diet is generally reflected in skin health, and changing from a poorly digestible generic diet to one with a better nutrient and digestive profile can often result in an improvement in the appearance of the hair coat and skin. The following subjects are considered: Essential fatty acid-responsive dermatoses; Retinoid-responsive dermatoses; Zinc-responsive dermatoses; Vitamin E dermatoses; and Superficial necrolytic dermatoses.

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28.2 Feline chin acne.
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28.3 Primary seborrhoea in a Cocker Spaniel.
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28.4 Zinc-responsive dermatosis. Periorbital crusting. Crusting on the ear margin. Nasal lesion. Nasal lesion following treatment.
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28.6 Superficial necrolytic dermatitis. Crusting and fissures on the footpads. Mucocutaneous crusting.
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28.7 Algorithm for working up a case of suspected superficial necrolytic dermatosis. = Most patients with SND have elevated liver enzymes; however, some dogs with end-stage liver disease may have normal liver enzyme concentrations. = Many cases of hepatic-induced SND (i.e. hepatocutaneous syndrome) are associated with chronic administration of hepatotoxic drugs such as phenobarbital. = Most dogs with hepatocutaneous syndrome have normal bile acids. However, if bile acids are elevated, the prognosis is significantly poorer.

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